Vascular and glomerular manifestations of viral hepatitis B and C: a review

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Secondary vasculopathies have varied etiologies that include paraneoplastic processes (eg, migratory thrombophlebitis, urticarial vasculitis); direct invasion by tumors (eg, superior and inferior vena cava syndrome); metabolic diseases (eg, diabetes mellitus); and infections, among others. The infective causes of vasculitides could result from direct involvement of vessels by a vasculo-tropic agent (eg, mucor infection); adjacent inflammation nonspecifically affecting nearby vessels; or from infection-induced immune-mediated vasculitis. Viruses represent a major group in the development of the latter, and many human viruses have been reported to cause vasculitis. The vasculitic lesions secondary to hepatitis B and hepatitis C viruses largely fall within the spectrum of immune-mediated secondary vascular injury and are discussed in this review.

Original languageEnglish (US)
Pages (from-to)116-121
Number of pages6
JournalSeminars in Diagnostic Pathology
Issue number2
StatePublished - May 1 2009
Externally publishedYes

Bibliographical note

Copyright 2010 Elsevier B.V., All rights reserved.


  • Cryoglobulinemia
  • Glomerulonephritis
  • HBV
  • HCV
  • Polyarteritis
  • Vasculitis
  • Vasculopathy
  • Viral hepatitis


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