Tyrosine phosphorylation of K_ir3.1 in spinal cord is induced by acute inflammation, chronic neuropathic pain, and behavioral stress

Keyphrases

• Spinal Cord 100%
• Chronic Neuropathic Pain 100%
• Tyrosine Phosphorylation 100%
• Behavioral Stress 100%
• Acute Inflammation 100%
• Kir3.1 100%
• Pain Stress 100%
• Kir3 37%
• Immunoreactivity 25%
• Spinal Nerve Ligation 25%
• Chronic Inflammatory Pain 25%
• Neuronal Excitability 12%
• Tyrosine 12%
• G Protein 12%
• Phosphorylation 12%
• Physiological Response 12%
• Targeted Gene Delivery 12%
• Channel Activity 12%
• Gene Expression Studies 12%
• Mammalian Cells 12%
• Inward Rectifier Potassium Channel 12%
• Transfected Cells 12%
• Primary Culture 12%
• Spinal Dorsal Horn 12%
• Dorsal Horn 12%
• N-terminal Domain 12%
• Cultured Human Cells 12%
• Kir3 Channels 12%
• Forced Swim Stress 12%
• Channel Conductance 12%
• Physiologically Relevant Conditions 12%
• Mouse Atrium 12%
• Basal Channels 12%
• Ion Channel Function 12%
• Xenopus Oocytes 12%
• Formalin Injection 12%
• Native State 12%
• Hind Paw 12%
• Superficial Layer 12%
• Deactivation 12%

Neuroscience

• Protein Phosphorylation 100%
• Neuropathic Pain 100%
• Sciatic Nerve 50%
• Ion Channel 25%
• G Protein 25%
• Neuronal Excitability 25%
• Primary Culture 25%
• Gene Expression 25%
• Cell Line 25%
• Amino Terminal Sequence 25%
• Inwardly Rectifying Potassium Channel 25%
• Gene Disruption 25%
• Spinal Cord Dorsal Horn 25%