Tyrosine phosphorylation of K_ir3.1 in spinal cord is induced by acute inflammation, chronic neuropathic pain, and behavioral stress

Keyphrases

• Spinal Cord 100%
• Chronic Neuropathic Pain 100%
• Tyrosine Phosphorylation 100%
• Behavioral Stress 100%
• Acute Inflammation 100%
• Pain Stress 100%
• Kir3 75%
• Immunoreactivity 50%
• Spinal Nerve Ligation 50%
• Chronic Inflammatory Pain 50%
• Neuronal Excitability 25%
• Tyrosine 25%
• Phosphorylation 25%
• Physiological Response 25%
• Targeted Gene Delivery 25%
• Channel Activity 25%
• Gene Expression Studies 25%
• Mammalian Cells 25%
• Transfected Cells 25%
• Primary Culture 25%
• Spinal Dorsal Horn 25%
• Dorsal Horn 25%
• N-terminal Domain 25%
• Kir3 Channels 25%
• Forced Swim Stress 25%
• Channel Conductance 25%
• Physiologically Relevant Conditions 25%
• Mouse Atrium 25%
• Ion Channel Function 25%
• Xenopus Oocytes 25%
• Formalin Injection 25%
• Native State 25%
• Hind Paw 25%
• Superficial Layer 25%
• Deactivation 25%
• G Protein-coupled Inwardly Rectifying Potassium Channels 25%
• Cultured Mammalian Cells 25%
• Basal Channel 25%

Neuroscience

• Spinal Cord 100%
• Protein Phosphorylation 100%
• Neuropathic Pain 100%
• Sciatic Nerve 50%
• Ion Channel 25%
• G Protein 25%
• Neuronal Excitability 25%
• Tyrosine 25%
• Primary Culture 25%
• Formaldehyde 25%
• Gene Expression 25%
• Cell Line 25%
• Amino Terminal Sequence 25%
• Inwardly Rectifying Potassium Channel 25%
• Gene Disruption 25%
• Spinal Cord Dorsal Horn 25%
• phosphorylation 25%