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The Role of Redox Cycling Versus Arylation in Quinone-Induced Mitochondrial Dysfunction: A Mechanistic Approach in Classifying Reactive Toxicants
T. R. Henry,
Kendall B Wallace
Biomedical Sciences
Research output
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Contribution to journal
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Article
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peer-review
6
Scopus citations
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Dive into the research topics of 'The Role of Redox Cycling Versus Arylation in Quinone-Induced Mitochondrial Dysfunction: A Mechanistic Approach in Classifying Reactive Toxicants'. Together they form a unique fingerprint.
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Keyphrases
Mitochondrial Dysfunction
100%
Toxicants
100%
Mechanistic Approach
100%
Quinones
100%
Redox Cycling
100%
1,4-naphthoquinone
100%
Arylation
100%
Cyclosporine
42%
1,4-benzoquinone
42%
Ca2+ Release
28%
Electrophilic
28%
Dimethyl
28%
Mitochondrial Permeability Transition
14%
Membrane Potential
14%
Membrane Depolarization
14%
Electrophilicity
14%
Stereoelectronic
14%
Induced Effect
14%
Calcium Flux
14%
One-electron Reduction Potential
14%
2-Hydroxy-1,4-naphthoquinone
14%
Quinone Toxicity
14%
Pharmacology, Toxicology and Pharmaceutical Science
Quinone Derivative
100%
1,4 Naphthoquinone
100%
Ciclosporin
50%
1,4 Benzoquinone
50%
Calcium Ion
33%
2,3 Dimethoxy 1,4 Naphthoquinone
16%
Menadione
16%