TY - JOUR
T1 - The influence of undertreated chronic pain in a national survey
T2 - Prescription medication misuse among American indians, Asian Pacific Islanders, Blacks, Hispanics and whites
AU - Johnson-Jennings, Michelle
AU - Duran, Bonnie
AU - Hakes, Jahn
AU - Paffrath, Alexandra
AU - Little, Meg M.
N1 - Publisher Copyright:
© 2020 The Authors
PY - 2020/8
Y1 - 2020/8
N2 - Objective: Disparities in the assessment and treatment of chronic pain among racial/ethnic may lead to self-treatment for undertreated pain. This study examines whether pain intensity among US racial/ethnic groups’ influences rates of psychotherapeutic prescription drug misuse. Methods: Data included civilian, non-institutionalized adults (age 18–99 years) residing in the United States (n = 34,653) from Waves 1 and 2 of the National Epidemiological Survey on Alcoholism and Related Conditions (NESARC; 2004–2005). The primary outcome variable was prescription drug misuse/PDM (i.e., use without a prescription or other than as prescribed) including tranquilizers, sedatives, stimulants, or opioids. Predictor variables included self-reported race/ethnicity (American Indian, Black, Hispanic, or White) and pain intensity. Data were analyzed in 2019. Results: Overall, White and Hispanic participants’ pain intensity had a significantly curvilinear relationship with frequency of prescription medication (p < 0.01). PDM rose with pain intensity until pain levels reached “severe,” then PDM rates fell, not significantly differing from the “no pain” levels (χ2(1) = 0.65, p = 0.42). PDM rates for Black participants remained lowest of all other racial/ethnic groups and plateaued with increasing pain intensity. Conclusions: Our results indicate that undertreated chronic pain may drive rates of PDM among varying racial/ethnic groups. Providing equitable assessment and treatment of pain intensity remains critical. Additional research is needed to examine provider decision-making and unconscious bias, as well as patient health beliefs surrounding perceived need for prescription pain medications.
AB - Objective: Disparities in the assessment and treatment of chronic pain among racial/ethnic may lead to self-treatment for undertreated pain. This study examines whether pain intensity among US racial/ethnic groups’ influences rates of psychotherapeutic prescription drug misuse. Methods: Data included civilian, non-institutionalized adults (age 18–99 years) residing in the United States (n = 34,653) from Waves 1 and 2 of the National Epidemiological Survey on Alcoholism and Related Conditions (NESARC; 2004–2005). The primary outcome variable was prescription drug misuse/PDM (i.e., use without a prescription or other than as prescribed) including tranquilizers, sedatives, stimulants, or opioids. Predictor variables included self-reported race/ethnicity (American Indian, Black, Hispanic, or White) and pain intensity. Data were analyzed in 2019. Results: Overall, White and Hispanic participants’ pain intensity had a significantly curvilinear relationship with frequency of prescription medication (p < 0.01). PDM rose with pain intensity until pain levels reached “severe,” then PDM rates fell, not significantly differing from the “no pain” levels (χ2(1) = 0.65, p = 0.42). PDM rates for Black participants remained lowest of all other racial/ethnic groups and plateaued with increasing pain intensity. Conclusions: Our results indicate that undertreated chronic pain may drive rates of PDM among varying racial/ethnic groups. Providing equitable assessment and treatment of pain intensity remains critical. Additional research is needed to examine provider decision-making and unconscious bias, as well as patient health beliefs surrounding perceived need for prescription pain medications.
KW - Chronic non cancer pain
KW - Prescription drug misuse
KW - US racial/Ethnic groups
KW - Under treated pain
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U2 - 10.1016/j.ssmph.2020.100563
DO - 10.1016/j.ssmph.2020.100563
M3 - Article
C2 - 32637551
AN - SCOPUS:85086872408
SN - 2352-8273
VL - 11
JO - SSM - Population Health
JF - SSM - Population Health
M1 - 100563
ER -