Susceptibility or resistance to high altitude pulmonary hypertension in cattle appears to be determined genetically, although the mechanism is obscure. Susceptible animals did not exhibit greater intrinsic reactivity of the pulmonary vasculature in response to prostaglandin F 2α. Neither did they have a greater hypoxic stimulus at altitude due to hypoventilation.
|Original language||English (US)|
|Number of pages||5|
|State||Published - Dec 1 1974|