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Spinocerebellar ataxia type 1
Harry T. Orr
Laboratory Medicine and Pathology
Genetics Mechanisms of Cancer
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Keyphrases
Adverse Effects
11%
Aggregated Protein
11%
Amino Acids
11%
Ataxia
11%
Ataxin-1
55%
Bulbar Dysfunction
11%
CAG Trinucleotide Repeat
11%
Cell Pathology
11%
Clinical Features
11%
Coding Region
11%
Disease Course
11%
Disease Mechanisms
11%
Disease Progression
11%
Disease Recovery
11%
Disease-causing mutations
11%
Dysarthria
11%
Gene Products
11%
Glutamine
22%
Inherited Ataxias
11%
Multiple System Atrophy
11%
Neurological Signs
11%
Neuropathology
11%
Pathophysiology
11%
Polyglutamine Expansion
11%
Polyglutamine Proteins
11%
Preclinical Animal Models
11%
Proteasomal Degradation
11%
Purkinje Cell
22%
Purkinje Neuron
11%
RNA Interference
22%
Spinocerebellar Ataxia
100%
Therapeutic Intervention
11%
Ubiquitin-proteasome System
11%
Ubiquitination
11%
Biochemistry, Genetics and Molecular Biology
Amino Acids
20%
Ataxin 1
100%
Coding Region
20%
Experimental Mouse
20%
Gene Product
20%
Glutamine
40%
Mouse Model
20%
Purkinje Cell
40%
RNA Interference
40%
Trinucleotide Repeat Expansion
20%
Ubiquitination
20%
Neuroscience
Adverse Effect
11%
Amino Acid
11%
Ataxia
22%
Ataxin 1
55%
Coding Region
11%
Dysarthria
11%
Gene Product
11%
Glutamine
22%
Multiple System Atrophy
11%
Polyglutamine
22%
Purkinje Cell
22%
Spinocerebellar Ataxia
100%
Trinucleotide Repeat
11%
Ubiquitination
11%