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Similar CD19 dysregulation in two autoantibody-associated autoimmune diseases suggests a shared mechanism of B-cell tolerance loss
Donna A. Culton
, Matilda W. Nicholas
, Donna O. Bunch
, Quan Li Zhen
, Thomas B. Kepler
, Mary Anne Dooley
, Chandra Mohan
,
Patrick Nachman
, Stephen H. Clarke
Research output
:
Contribution to journal
›
Article
›
peer-review
61
Scopus citations
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Dive into the research topics of 'Similar CD19 dysregulation in two autoantibody-associated autoimmune diseases suggests a shared mechanism of B-cell tolerance loss'. Together they form a unique fingerprint.
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Keyphrases
Antigen Selection
25%
Antineutrophil Cytoplasmic Antibody
75%
Array Analysis
25%
Autoantibodies
100%
Autoantibody Profile
25%
Autoantigen
25%
Autoreactive B Cells
25%
B Cell Activation
25%
B Cell Tolerance
100%
B Cells
100%
B-cell Receptor Signaling
25%
CD19
100%
Co-receptor
25%
Dysregulation
100%
Healthy Controls
50%
Large Vessel Vasculitis
75%
Memory B Cells
50%
Nave
25%
Proteome Array
25%
Secondary Autoimmune Disease
100%
Small nuclear Ribonucleoprotein (snRNP)
25%
Systemic Lupus Erythematosus
100%
Immunology and Microbiology
Autoantibodies
100%
Autoantigen
16%
Autoimmune Disease
100%
B Cell
66%
B Cell Activation
16%
B-Cell Receptor
16%
B-Cell Tolerance
100%
CD19
100%
Memory B Cell
33%
Systemic Lupus Erythematosus
66%
Vasculitis
50%