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SH2B3 aberrations enriched in iAMP21 B lymphoblastic leukemia
L. B. Baughn
, M. M. Meredith
, L. Oseth
, T. A. Smolarek
,
B. Hirsch
Laboratory Medicine and Pathology
Masonic Cancer Center
Research output
:
Contribution to journal
›
Article
›
peer-review
20
Scopus citations
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Keyphrases
Aberrations
100%
SH2B3
100%
B-lymphoblastic Leukemia
100%
Intrachromosomal Amplification of Chromosome 21 (iAMP21)
100%
Leukemia
75%
Poor Prognosis
50%
B-cell Acute Lymphoblastic Leukemia (B-ALL)
50%
Fluorescence in Situ Hybridization
50%
Chromosomal Microarray
50%
Disease Progression
25%
Pediatric
25%
High Risk
25%
Tyrosine Kinase Inhibitor
25%
Acute Lymphoblastic Leukemia
25%
Negative Regulator
25%
Therapeutic Intervention
25%
Biological Mechanisms
25%
Nucleotide Variation
25%
Next-generation Sequencing
25%
Cytokine Signaling
25%
Loss of Heterozygosity
25%
Genetic Aberrations
25%
Childhood Malignancy
25%
Chromosome 21
25%
Copy number Aberrations
25%
G-banding
25%
Inpatient Outcomes
25%
RUNX1 Gene
25%
Tyrosine Kinase Signaling Pathway
25%
Biochemistry, Genetics and Molecular Biology
SH2B3
100%
Chromosome 21
100%
Fluorescence in Situ Hybridization
40%
Genetics
20%
Tyrosine Kinase Inhibitor
20%
Tyrosine Kinase
20%
Loss of Heterozygosity
20%
Next Generation Sequencing
20%
G Banding
20%
RUNX1
20%
Cytokine
20%
Receptor Tyrosine Kinase
20%