Redundancy in antigen-presenting function of the HLA-DR and -DQ molecules in the multiple sclerosis-associated HLA-DR2 haplotype

Keyphrases

• Multiple Sclerosis 100%
• Redundancy 100%
• Antigen-presenting 100%
• HLA-DR 100%
• HLA-DR2 100%
• Disease-associated 66%
• T Cell Clone 66%
• HLA Class II Alleles 66%
• CD4+ T 33%
• Autoimmune Disease 33%
• T Cell Activation 33%
• T Cell Signaling 33%
• Linkage Disequilibrium 33%
• Cell-mediated 33%
• Cerebrospinal Fluid 33%
• Ag Presentation 33%
• Structural Features 33%
• B Cell Lines 33%
• Genetic Risk 33%
• DRB1 33%
• HLA Class II 33%
• Multiple Diseases 33%
• Functional Redundancy 33%
• HLA Haplotypes 33%
• Infiltrating T Cells 33%
• HLA-DQB1 33%
• Combinatorial Peptide Library 33%
• HLA-peptide Complex 33%

Immunology and Microbiology

• Multiple Sclerosis 100%
• HLA-DR 100%
• Haplotype 100%
• T Cell Receptor 50%
• Cell Clone 50%
• T Cell 50%
• T-Helper Cell 25%
• B Cell 25%
• Cell Line 25%
• T Cell Activation 25%
• T Cell-Mediated Autoimmune Disease 25%
• T-Cell Signaling 25%
• Genetic Risk 25%
• Peptide Library 25%
• Gene Linkage Disequilibrium 25%
• Cerebrospinal Fluid 25%
• Allele 25%
• Comorbidity 25%

Biochemistry, Genetics and Molecular Biology

• Haplotype 100%
• HLA-DR 100%
• HLA-DR2 100%
• T Cell 100%
• T Cell Receptor 40%
• Cell Clone 40%
• Signal Transduction 20%
• Liquid 20%
• Allele 20%
• Genetic Risk 20%
• Gene Linkage Disequilibrium 20%
• Cell Activation 20%
• Peptide Library 20%
• B Cell 20%
• CD4 20%
• Comorbidity 20%