Progesterone inhibition of voltage-gated calcium channels is a potential neuroprotective mechanism against excitotoxicity

Keyphrases

• Activity-dependent Gene Expression 20%
• AMPA Receptor (AMPAR) 10%
• Beneficial Effects 10%
• Calcium Entry 10%
• Calcium Imaging 10%
• Calcium Overload 10%
• Calcium Signaling 10%
• Cell Behavior 10%
• Change of Measure 10%
• Depolarization 10%
• Electrophysiology 10%
• Excitotoxic 10%
• Far-reaching Effect 10%
• Neuroprotectant 10%
• Neuroprotection 10%
• Neuroprotective 10%
• Neuroprotective Mechanisms 100%
• Neurotoxicity 100%
• NMDA Signaling 10%
• Phase III Clinical Trial 10%
• Progesterone 100%
• Receptor-mediated 10%
• Shock 10%
• Steroid Hormones 10%
• Striatal Neurons 10%
• Therapeutic Concentrations 10%
• Therapeutic Use 10%
• Transcription Factor 10%
• Traumatic Brain Injury 10%
• Voltage-gated Calcium Channels 100%

Neuroscience

• AMPA Receptor 10%
• Calcium Imaging 10%
• Calcium Signaling 10%
• Cell Function 10%
• CREB 10%
• Electrophysiology 10%
• Excitotoxicity 100%
• Gene Expression 10%
• Neuroprotection 10%
• Neuroprotective Agent 100%
• Neurotrauma 10%
• NMDA Receptor 10%
• Progesterone 100%
• Receptor 10%
• Steroid Hormone 10%
• Transcription Factor NFAT 10%
• Traumatic Brain Injury 10%
• Voltage Gated Calcium Channel 100%