Neurochemical alterations in spinocerebellar ataxia type 1 and their correlations with clinical status

Keyphrases

• Spinocerebellar Ataxia 100%
• Neurochemicals 100%
• Clinical Status 100%
• Myo-inositol 100%
• Glutamate 60%
• Pons 60%
• 1H-MRS 40%
• Ataxia 40%
• Glutamine 40%
• Cerebellar Hemisphere 40%
• F2-isoprostanes 20%
• Tesla 20%
• Imaging Biomarkers 20%
• High Field 20%
• Cerebellum 20%
• Energy Metabolism 20%
• Brainstem 20%
• Oxidative Stress Markers 20%
• Cerebrospinal Fluid 20%
• Healthy Controls 20%
• Neuronal Dysfunction 20%
• Neurochemical Profile 20%
• Proton Magnetic Resonance Spectroscopy 20%
• Patient Relatives 20%
• Glial Fibrillary Acidic Protein 20%
• N-acetylaspartate 20%
• Clinical Applicability 20%
• Phosphocreatine 20%
• N-acetylaspartylglutamate 20%
• N-15 20%
• Gliosis 20%
• Vermis 20%
• Robust Biomarker 20%
• Ataxia Score 20%
• Score Scale 20%
• Neuroprotective Therapy 20%
• Metabolite Concentrations 20%

Neuroscience

• Inositol 100%
• Spinocerebellar Ataxia 100%
• Glutamic Acid 80%
• Pons 60%
• Ataxia 60%
• Glutamine 40%
• Cerebellar Hemisphere 40%
• Neuroprotective Agent 20%
• Brainstem 20%
• Glial Fibrillary Acidic Protein 20%
• Nuclear Magnetic Resonance Spectroscopy 20%
• Cerebellum 20%
• Isoprostane 20%
• Oxidative Stress 20%
• Creatine Phosphate 20%
• Energy Metabolism 20%
• Neurodegeneration 20%
• Gliosis 20%