Myocardial Upregulation of Cathepsin D by Ischemic Heart Disease Promotes Autophagic Flux and Protects Against Cardiac Remodeling and Heart Failure

Keyphrases

• Heart Failure 100%
• Cardiac Remodeling 100%
• Ischemic Heart Disease 100%
• Cathepsin D 100%
• Autophagic Flux 100%
• Myocardial Infarction 75%
• Human Heart Failure 50%
• Fractional Shortening 50%
• Failing Heart 25%
• Lysosomal Disease 25%
• Autophagosome 25%
• Left Ventricle 25%
• Lysosomal Acid Lipase 25%
• Cardiac Output 25%
• Western Blot Analysis 25%
• Ejection Fraction 25%
• Stroke Volume 25%
• Diastolic Function 25%
• Systolic Function 25%
• Mature Form 25%
• Wall Thinning 25%
• Scar Size 25%
• Pressure-volume Relationship 25%
• Dilatation 25%
• Restrictive Cardiomyopathy 25%
• Stroke Work 25%
• Coronary Artery Ligation 25%
• Lung Weight 25%
• Body Weight Ratio 25%
• Relationship Analysis 25%
• Serial Echocardiography 25%
• Chronic Myocardial Infarction 25%

Medicine and Dentistry

• Ischemic Heart Disease 100%
• Myocardial Infarction 100%
• Heart Ventricle Remodeling 100%
• Congestive Heart Failure 100%
• Upregulation 100%
• Cathepsin D 100%
• Body Weight 50%
• Apoplexy 25%
• Lung 25%
• Precursor 25%
• Ejection Fraction 25%
• Left Ventricle 25%
• Echocardiography 25%
• Stroke Volume 25%
• Diastolic Function 25%
• Systolic Function 25%
• Proteinase 25%
• Restrictive Cardiomyopathy 25%
• Heart Output 25%
• Western Blot 25%
• Coronary Artery Ligation 25%
• Autophagosome 25%

Pharmacology, Toxicology and Pharmaceutical Science

• Heart Infarction 100%
• Heart Ventricle Remodeling 100%
• Congestive Heart Failure 100%
• Cathepsin D 100%
• Ischemic Heart Disease 100%
• Cerebrovascular Accident 50%
• Western Blot 25%
• Proteinase 25%
• Restrictive Cardiomyopathy 25%