A long-term laboratory exposure of lake trout to Lake Ontario sediment and smelt (food chain) provided comprehensive bioaccumulation relationships for 2,3,7,8-TCDD. The laboratory exposure was designed to investigate the rates of TCDD uptake via water, sediment, and food under simulated Lake Ontario conditions. Innovative methods of preparing sediment, dosing sediment, preparing food and feeding the fish were developed. Results indicated that bioaccumulation of 2,3,7,8-TCDD occurs primarily through the food chain and secondarily through contact with contaminated sediment. The water exposure route, even under simulated equilibrium conditions, and low suspended solids concentrations did not appear to make a significant contribution to 2,3,7,8-TCDD bioaccumulation.
|Original language||English (US)|
|Number of pages||8|
|State||Published - 1989|
Bibliographical noteFunding Information:
The efforts of Douglas Kuehl of the Environmental Research Laboratory-Duluth (ERL-D) in giving guidance on analytical methods and assistance with HRGC/MS runs is greatly appreciated. We are also indebted to David Marklund, Sarah Kohlbry, Christine Harper, and John Libal of the Natural Resources Research Institute (NRRI) for dioxin analysis and general technical assistance and to Jim Gordon, NRRI, and David Michaelson of ERL-D for fish exposure support. The assistance of Patrick O'Keefe, New York Department of Health in furnishing quality assurance samples is appreciated. Appreciation is also extended to Russell Short, Ecology and Environment, Buffalo, New York, and Floyd Boettcher of ERL-D for field sampling efforts, and to the Gradient Corporation, Cambridge, Massachusetts for the overall coordination of the Lake Ontario TCDD Bioaccumulation Study. A major portion of this research was conducted by NRR\[ with the support of EPA cooperative agreement CR 813504-01. Mention of trade names or commercial products does not constitute endorsement or recommendations for use.
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- Lake Ontario
- food chain
- lake trout