Involvement of inducible 6-phosphofructo-2-kinase in the anti-diabetic effect of peroxisome proliferator-activated receptor γ activation in mice

Keyphrases

• Peroxisome Proliferator-activated Receptor 100%
• Receptor Activation 100%
• 6-phosphofructo-2-kinase 100%
• PFKFB3 100%
• Antidiabetic Effect 100%
• Adipocyte 30%
• Knockdown 30%
• Insulin Resistance 20%
• Glucose Intolerance 20%
• High-fat Diet 20%
• Diet-induced 20%
• Inflammatory Pathways 20%
• Insulin Signaling 20%
• Inflammatory Cytokine Response 20%
• Hyperglycemia 10%
• Fatty Acid Oxidation 10%
• Adipose Tissue 10%
• Littermate 10%
• Glycolysis 10%
• 3T3-L1 Adipocytes 10%
• Regulatory Enzymes 10%
• Lipid Accumulation 10%
• Rosiglitazone 10%
• Euglycemia 10%
• Thiazolidinediones 10%
• Adipocyte Lipolysis 10%
• Adipokine Expression 10%
• Visceral Fat Accumulation 10%

Biochemistry, Genetics and Molecular Biology

• 6-Phosphofructo-2-Kinase 100%
• Antidiabetic Activity 100%
• Peroxisome Proliferator-Activated Receptor 100%
• Adipocyte 57%
• Insulin Signaling 28%
• Lipid Storage 28%
• Proinflammatory Cytokine 28%
• Insulin Resistance 28%
• Isoform 14%
• Beta Oxidation 14%
• Wild Type 14%
• Lipolysis 14%
• Regulatory Enzymes 14%
• Hyperglycemia 14%
• 3T3-L1 14%
• Cytokine Production 14%
• Rosiglitazone 14%
• Adipokine 14%
• Thiazolidinedione 14%

Immunology and Microbiology

• Antidiabetic Activity 100%
• Peroxisome 100%
• Adipocyte 57%
• Adipose Tissue 28%
• Proinflammatory Cytokine 28%
• Lipid Storage 28%
• Insulin Signaling 28%
• Insulin Resistance 28%
• Adipokine 14%
• Cytokine Production 14%
• Fatty Acid Oxidation 14%
• Wild Type 14%
• Lipolysis 14%