Intimate partner violence in late adolescence and young adulthood and subsequent cardiovascular risk in adulthood

Cari J Clark, Alvaro Alonso, Susan Everson-Rose, Rachael A. Spencer, Sonya S Brady, Michael D Resnick, Iris W Borowsky, John E Connett, Robert Krueger, Viann Nguyen-Feng, Steven L. Feng, Shakira F. Suglia

Research output: Contribution to journalArticle

15 Citations (Scopus)

Abstract

Background: Childhood maltreatment has been linked to adulthood cardiovascular disease (CVD). Little is known about the relationship between intimate partner violence (IPV) in late adolescence and young adulthood and CVD risk later in adulthood. Purpose: To examine whether IPV perpetration and victimization experienced in late adolescence and young adulthood are associated with CVD risk among adults in the United States and whether this relationship differs by sex. Methods: Data include 9976 participants (50% female) in the National Longitudinal Study of Adolescent to Adult Health. Physical and sexual IPV were measured at wave 3 (2001/02) with items from the revised Conflict Tactics Scales. Participants'30-year risk of CVD was calculated at wave 4 (2008/09) using a Framingham prediction model. Linear regression models adjusted for confounders and IPV by sex interaction terms were tested to examine the relationship. Results: The mean CVD risk score was 13.18% (95% CI: 12.71, 13.64). Aone-standard deviation increase in the victimization score was associated with a 0.28% (95% CI: 0.03, 0.54) increase in CVD risk. Perpetration was similarly positively associated with CVD risk (beta: 0.33, 95% CI: 0.03, 0.62). When measured as a composite, all violence types were associated with increased CVD risk but only prior exposure to both victimization and perpetration reached statistical significance (0.62%, 95% CI: 0.01, 1.22). No differences by sex were detected. Conclusions: Effect sizes are not large, but early detection of increased CVD risk in this relatively young population is notable and worthy of further study to inform the clinical response.

Original languageEnglish (US)
Pages (from-to)132-137
Number of pages6
JournalPreventive Medicine
Volume87
DOIs
StatePublished - Jun 1 2016

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Cardiovascular Diseases
Crime Victims
Linear Models
Intimate Partner Violence
Sexual Partners
Violence
Sex Characteristics
Longitudinal Studies
Health
Population

Keywords

  • Adolescent health services
  • Cardiovascular diseases
  • Domestic violence
  • National Longitudinal Study of Adolescent Health

Cite this

Intimate partner violence in late adolescence and young adulthood and subsequent cardiovascular risk in adulthood. / Clark, Cari J; Alonso, Alvaro; Everson-Rose, Susan; Spencer, Rachael A.; Brady, Sonya S; Resnick, Michael D; Borowsky, Iris W; Connett, John E; Krueger, Robert; Nguyen-Feng, Viann; Feng, Steven L.; Suglia, Shakira F.

In: Preventive Medicine, Vol. 87, 01.06.2016, p. 132-137.

Research output: Contribution to journalArticle

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title = "Intimate partner violence in late adolescence and young adulthood and subsequent cardiovascular risk in adulthood",
abstract = "Background: Childhood maltreatment has been linked to adulthood cardiovascular disease (CVD). Little is known about the relationship between intimate partner violence (IPV) in late adolescence and young adulthood and CVD risk later in adulthood. Purpose: To examine whether IPV perpetration and victimization experienced in late adolescence and young adulthood are associated with CVD risk among adults in the United States and whether this relationship differs by sex. Methods: Data include 9976 participants (50{\%} female) in the National Longitudinal Study of Adolescent to Adult Health. Physical and sexual IPV were measured at wave 3 (2001/02) with items from the revised Conflict Tactics Scales. Participants'30-year risk of CVD was calculated at wave 4 (2008/09) using a Framingham prediction model. Linear regression models adjusted for confounders and IPV by sex interaction terms were tested to examine the relationship. Results: The mean CVD risk score was 13.18{\%} (95{\%} CI: 12.71, 13.64). Aone-standard deviation increase in the victimization score was associated with a 0.28{\%} (95{\%} CI: 0.03, 0.54) increase in CVD risk. Perpetration was similarly positively associated with CVD risk (beta: 0.33, 95{\%} CI: 0.03, 0.62). When measured as a composite, all violence types were associated with increased CVD risk but only prior exposure to both victimization and perpetration reached statistical significance (0.62{\%}, 95{\%} CI: 0.01, 1.22). No differences by sex were detected. Conclusions: Effect sizes are not large, but early detection of increased CVD risk in this relatively young population is notable and worthy of further study to inform the clinical response.",
keywords = "Adolescent health services, Cardiovascular diseases, Domestic violence, National Longitudinal Study of Adolescent Health",
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AU - Clark, Cari J

AU - Alonso, Alvaro

AU - Everson-Rose, Susan

AU - Spencer, Rachael A.

AU - Brady, Sonya S

AU - Resnick, Michael D

AU - Borowsky, Iris W

AU - Connett, John E

AU - Krueger, Robert

AU - Nguyen-Feng, Viann

AU - Feng, Steven L.

AU - Suglia, Shakira F.

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N2 - Background: Childhood maltreatment has been linked to adulthood cardiovascular disease (CVD). Little is known about the relationship between intimate partner violence (IPV) in late adolescence and young adulthood and CVD risk later in adulthood. Purpose: To examine whether IPV perpetration and victimization experienced in late adolescence and young adulthood are associated with CVD risk among adults in the United States and whether this relationship differs by sex. Methods: Data include 9976 participants (50% female) in the National Longitudinal Study of Adolescent to Adult Health. Physical and sexual IPV were measured at wave 3 (2001/02) with items from the revised Conflict Tactics Scales. Participants'30-year risk of CVD was calculated at wave 4 (2008/09) using a Framingham prediction model. Linear regression models adjusted for confounders and IPV by sex interaction terms were tested to examine the relationship. Results: The mean CVD risk score was 13.18% (95% CI: 12.71, 13.64). Aone-standard deviation increase in the victimization score was associated with a 0.28% (95% CI: 0.03, 0.54) increase in CVD risk. Perpetration was similarly positively associated with CVD risk (beta: 0.33, 95% CI: 0.03, 0.62). When measured as a composite, all violence types were associated with increased CVD risk but only prior exposure to both victimization and perpetration reached statistical significance (0.62%, 95% CI: 0.01, 1.22). No differences by sex were detected. Conclusions: Effect sizes are not large, but early detection of increased CVD risk in this relatively young population is notable and worthy of further study to inform the clinical response.

AB - Background: Childhood maltreatment has been linked to adulthood cardiovascular disease (CVD). Little is known about the relationship between intimate partner violence (IPV) in late adolescence and young adulthood and CVD risk later in adulthood. Purpose: To examine whether IPV perpetration and victimization experienced in late adolescence and young adulthood are associated with CVD risk among adults in the United States and whether this relationship differs by sex. Methods: Data include 9976 participants (50% female) in the National Longitudinal Study of Adolescent to Adult Health. Physical and sexual IPV were measured at wave 3 (2001/02) with items from the revised Conflict Tactics Scales. Participants'30-year risk of CVD was calculated at wave 4 (2008/09) using a Framingham prediction model. Linear regression models adjusted for confounders and IPV by sex interaction terms were tested to examine the relationship. Results: The mean CVD risk score was 13.18% (95% CI: 12.71, 13.64). Aone-standard deviation increase in the victimization score was associated with a 0.28% (95% CI: 0.03, 0.54) increase in CVD risk. Perpetration was similarly positively associated with CVD risk (beta: 0.33, 95% CI: 0.03, 0.62). When measured as a composite, all violence types were associated with increased CVD risk but only prior exposure to both victimization and perpetration reached statistical significance (0.62%, 95% CI: 0.01, 1.22). No differences by sex were detected. Conclusions: Effect sizes are not large, but early detection of increased CVD risk in this relatively young population is notable and worthy of further study to inform the clinical response.

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