Abstract
Carcinogenesis is influenced by numerous factors, including a tissue microenvironment that is associated with chronic inflammation. While acute inflammation is a physiological response to tissue damage or infection with the goal of restoring tissue homeostasis, chronic inflammation can contribute to a variety of pathological conditions, including cancer. This review explores the complex relationship between inflammation and carcinogenesis, examining the mechanisms through which inflammation contributes to cancer initiation and progression by focusing on the roles of key inflammatory cells and inflammatory mediators. Ongoing efforts to target inflammation to reduce cancer risk and reduce cancer progression are also discussed.
| Original language | English (US) |
|---|---|
| Title of host publication | Comprehensive Toxicology |
| Publisher | Elsevier |
| Pages | Vol13:536-Vol15:555 |
| ISBN (Electronic) | 9780323954884 |
| ISBN (Print) | 9780323954891 |
| DOIs | |
| State | Published - Jan 1 2025 |
Bibliographical note
Publisher Copyright:© 2026 Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
UN SDGs
This output contributes to the following UN Sustainable Development Goals (SDGs)
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SDG 3 Good Health and Well-being
Keywords
- Carcinogenesis
- Chemokines
- Cytokines
- Fibroblast
- Immunity
- Immunotherapy
- Inflammation
- Interferons
- Interleukins
- Macrophage
- Neutrophil
- Tumor microenvironment
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