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IFN-γ signaling stimulates intestinal crypt hyperplasia in celiac disease
Alexa R. Weingarden
Medicine - Gastro, Hepatology, Nutrition Division
Research output
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Contribution to journal
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Article
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peer-review
Overview
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Keyphrases
Celiac Disease
100%
IFN Signaling
100%
Intestinal Crypts
100%
Crypt Hyperplasia
100%
Villi
66%
Crypt Cells
66%
Protein Expression
33%
Immune Response
33%
Fatty Acid Metabolism
33%
Enteropathy
33%
Epithelial Cells
33%
Expression Pattern
33%
Intestinal Epithelial Cells
33%
Decreased Expression
33%
Compensatory Response
33%
IFN Response
33%
Metabolic Pathways
33%
Dietary Gluten
33%
IFN Receptor
33%
Medicine and Dentistry
Coeliac Disease
100%
Hyperplasia
100%
Crypt
100%
Crypt Cell
66%
Immune Response
33%
Receptor
33%
Epithelial Cell
33%
Fatty Acid Metabolism
33%
Enteropathy
33%
Immunology and Microbiology
Coeliac Disease
100%
Crypt Cell
66%
Fatty Acid Metabolism
33%
Immune Response
33%
Biochemistry, Genetics and Molecular Biology
Fatty Acid Metabolism
100%
Immune Response
100%