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Expression of human PKR protein kinase in transgenic mice
Warren Ladiges
, John Morton
, Heather Hopkins
, Ruth Wilson
, Gina Filley
, Carol Ware
,
Michael Gale
Research output
:
Contribution to journal
›
Article
›
peer-review
5
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Scopus citations
Overview
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Keyphrases
Transgenic Mice
100%
Protein Kinase
100%
Null Mice
66%
Spleen Cells
66%
Cell Growth
33%
Overexpressing
33%
Redundancy
33%
Tumor Suppressor
33%
In Vitro Study
33%
Scientific Disciplines
33%
Apoptosis
33%
Interferon-α (IFN-α)
33%
Growth Regulators
33%
Innate Resistance
33%
Actinomycin D
33%
Genotoxic Agents
33%
Transcription-translation
33%
Double-stranded RNA
33%
Double-stranded RNA-dependent Protein Kinase
33%
Protein Kinase PKR
33%
Small Bodies
33%
Regulatory Pathways
33%
Transgenic Mouse Lines
33%
Resistance to Viral Infection
33%
Canavalin
33%
Biochemistry, Genetics and Molecular Biology
Transgenic Mouse
100%
Protein Kinase R
100%
Wild Type
50%
Knockout Mouse
50%
Phosphotransferase
50%
Kinase
50%
Splenocyte
50%
Tumor Suppressor Protein
25%
Transcription
25%
In Vitro Study
25%
Double-Stranded RNA
25%
Cell Growth
25%
Dactinomycin
25%
Interferon
25%
Programmed Cell Death
25%
Immunology and Microbiology
Transgenic Mouse
100%
Splenocyte
50%
Knockout Mouse
50%
Wild Type
50%
Double-Stranded RNA
50%
Viral Disease
25%
Biological Product
25%
Human Interferon
25%
Cell Growth
25%
Dactinomycin
25%
Programmed Cell Death
25%
In Vitro
25%
Pharmacology, Toxicology and Pharmaceutical Science
Transgenic Mouse
100%
Protein Kinase R
100%
Knockout Mouse
50%
Phosphotransferase
50%
Dactinomycin
25%
Tumor Suppressor Protein
25%
Virus Infection
25%
Biological Product
25%
Human Interferon
25%
Diseases
25%