Evidence for a key role of steroids in the modulation of pain

Keyphrases

• Anti-inflammatory Properties 50%
• Calcitonin Gene-related Peptide 50%
• Chemical Characteristics 50%
• Chronic Pain 50%
• Development Activities 50%
• Development Plasticity 50%
• Dimorphism 50%
• Eicosanoids 50%
• Endogenous Corticosteroids 50%
• Endogenous Production 50%
• Endogenous Steroids 50%
• Free Radicals 50%
• Glutamate 50%
• Nervous System 50%
• Neural Circuits 50%
• Neurobiological Processes 50%
• Neuropathic Pain 50%
• Neurosteroids 50%
• Neurotransmitters 50%
• Pain Mechanisms 50%
• Pain Perception 100%
• Pain Sensation 100%
• Pain Transmission 50%
• Perceptual Effects 50%
• Regulatory Effect 100%
• Second Category 50%
• Serotonin 50%
• Sex Steroids 100%
• Spinal Cord 50%
• Steroid-induced 50%
• Steroidal Compounds 50%
• Substance P 50%
• Synthetic Steroids 50%
• Transcription Factor 50%
• β-endorphin 50%
• γ-aminobutyric Acid 50%

Neuroscience

• Anti-Inflammatory 12%
• Calcitonin Gene Related Peptide 12%
• Calcitonin Gene-Related Peptide 12%
• Cytokine 12%
• Eicosanoid 12%
• Endorphin 12%
• Free Radical 12%
• Gamma-Aminobutyric Acid 12%
• Glutamic Acid 12%
• Nervous System 12%
• Neural Circuit 12%
• Neuropathic Pain 12%
• Neurosteroid 25%
• Neurotransmitter 12%
• Nociception 37%
• Sensation of Pain 12%
• Serotonin 12%
• Sex Hormone 12%
• Spinal Cord 12%
• Steroids 100%
• Substance P 12%
• Transcription Factors 12%