Ethylene signaling pathway and MAPK cascades are required for AAL toxin-induced Programmed cell death

Keyphrases

• Protein Kinase 100%
• Programmed Cell Death 100%
• Ethylene Signaling 100%
• MAPK Cascade 100%
• AAL-toxin 100%
• Acid Activation 50%
• Salicylic Acid 50%
• Wound-induced 50%
• MEK2 40%
• Cell Death 30%
• Mitogen-activated Protein Kinase 20%
• Virus-induced Gene Silencing 20%
• Ethylene Treatment 20%
• Signaling Pathway 10%
• Ethylene 10%
• Tobacco Rattle Virus 10%
• Pathogenicity Factors 10%
• Kinase Cascade 10%
• Hypersensitive Cell Death 10%
• Plant Programmed Cell Death 10%
• Plant Defense Response 10%
• Necrotrophic Pathogen 10%
• Ethylene Synthesis 10%
• Ethylene Biosynthesis 10%
• Alternaria Alternata 10%
• Lycopersici 10%
• Ethylene Accumulation 10%
• Nicotiana 10%

Biochemistry, Genetics and Molecular Biology

• Signal Transduction 100%
• Cell Death 100%
• Mitogen-Activated Protein Kinase 100%
• Protein Kinases 100%
• Ethylene 100%
• Infectious Agent 20%
• Gene Inactivation 20%
• Anabolism 10%
• Defense Response 10%
• Plant Defense 10%
• Nicotiana 10%
• Tobacco Rattle Virus 10%
• Mitogen-Activated Protein Kinase Kinase 10%
• Alternaria alternata 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Programmed Cell Death 100%
• Ethylene 100%
• Protein Kinases 100%
• Salicylic Acid 50%
• Wound 50%
• Infectious Agent 20%
• Pathogenicity 10%
• Mitogen Activated Protein Kinase 10%
• Mitogen Activated Protein Kinase Kinase 10%
• Allergic Reaction 10%
• Nicotiana 10%
• Tobacco Rattle Virus 10%
• Alternaria alternata 10%