Elucidating mechanisms of genetic cross-disease associations at the PROCR vascular disease locus

Keyphrases

• Vascular Disease 100%
• Disease Association 100%
• Procr 100%
• Cross-disease 100%
• Genetic Crosses 100%
• Endothelial Protein C Receptor 33%
• Causal Variants 16%
• Genetic Analysis 16%
• Endothelial Cells 16%
• Molecular Mechanism 16%
• Coronary Artery Disease 16%
• Venous Thromboembolism 16%
• Coronary Risk Factors 16%
• Human Disease 16%
• Causal Factors 16%
• Cell Adhesion 16%
• Integrative Approach 16%
• Genotype-phenotype Association 16%
• Coagulation Factor VII 16%
• Plasma Parameters 16%
• Thrombotic 16%
• Pleiotropy 16%
• Vascular Inflammation 16%
• Prothrombotic State 16%
• Ectodomain Shedding 16%
• Activated Protein C 16%
• In Vitro Experimentation 16%
• Anti-inflammatory Mechanism 16%
• Genetic Risk Loci 16%
• Recall-by-genotype 16%
• Venous Thromboembolism Risk 16%

Biochemistry, Genetics and Molecular Biology

• Genetic Cross 100%
• Endothelial Protein C Receptor 100%
• Protein C 100%
• Genetics 50%
• Cell Adhesion 50%
• Genotyping 50%
• Pleiotropy 50%
• Ectodomain 50%
• Genetic Risk 50%
• Blood Level 50%
• Factor VII 50%