Effects of caspase inhibitor on angiotensin II-induced abdominal aortic aneurysm in apolipoprotein E-deficient mice

Keyphrases

• Ruptured Abdominal Aortic Aneurysm (rAAA) 100%
• Angiotensin II (Ang II) 100%
• Apolipoprotein E Knockout Mice 100%
• Caspase Inhibitor 100%
• Quinoline 75%
• Monocyte Chemoattractant protein-1 (MCP-1) 50%
• Apoptosis 50%
• Conditioned Medium 50%
• Angiotensin II Infusion 50%
• Aneurysm Formation 50%
• Inflammation 25%
• Neutralizing Antibodies 25%
• Macrophages 25%
• Smooth muscle Cells 25%
• Vascular Disease 25%
• Aneurysm 25%
• Aortic Diameter 25%
• Cell Apoptosis 25%
• Pan-caspase Inhibitor 25%
• Vascular Inflammation 25%
• Caspase-dependent 25%
• Apoptotic Markers 25%
• Smooth muscle Cell Apoptosis 25%
• Macrophage Chemotaxis 25%
• Aneurysm Development 25%
• Histopathological Features 25%

Neuroscience

• Caspase 100%
• Angiotensin II 100%
• Apolipoprotein E 100%
• Aneurysm 100%
• Programmed Cell Death 66%
• Macrophage 33%
• Monocyte Chemotactic Protein 1 33%
• Vascular Disease 16%
• In Vitro 16%
• Chemotaxis 16%
• Neutralizing Antibody 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Abdominal Aortic Aneurysm 100%
• Angiotensin II 100%
• Caspase Inhibitor 100%
• Apolipoprotein E 100%
• Aneurysm 66%
• Monocyte Chemotactic Protein 1 33%
• Inflammation 16%
• Caspase 16%
• Neutralizing Antibody 16%
• Vasculitis 16%
• Vascular Disease 16%