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Effect of intravenous silica on the course of Nocardia asteroides pneumonia

Research output: Contribution to journalArticlepeer-review

Abstract

Silica, a known toxin of mononuclear phagocytes, was administered intravenously to mice during Nocardia asteroides pneumonia. Mice that received silica had a sevenfold decrease in the number of peripheral blood monocytes and developed more severe N. asteroides pneumonia than control mice. Lung histology in mice that received silica resembled that of mice with impaired cell-mediated immunity. These results are most consisent with the explanation that silica injures blood monocytes and impaires their contributions to pulmonary host defense.

Original languageEnglish (US)
Pages (from-to)2507-2511
Number of pages5
JournalInfection and immunity
Volume56
Issue number9
StatePublished - Jan 1 1988

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

  1. SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well-being

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