Differential estrogen-receptor activation regulates extracellular matrix deposition in human airway smooth muscle remodeling via NF-κB pathway

Keyphrases

• Estrogen Receptor 100%
• Human Airway Smooth muscle 100%
• Receptor Activation 100%
• NF-B 100%
• Extracellular Matrix Deposition 100%
• Smooth muscle Remodeling 100%
• Platelet-derived Growth factor-BB (PDGF-BB) 25%
• Airway Remodeling 16%
• Asthma 16%
• Extracellular Matrix 16%
• Airway Smooth muscle 16%
• Activator protein-1 (AP-1) 16%
• Non-asthmatics 16%
• Asthmatic 16%
• Estrogen Receptor Agonists 16%
• Airway Smooth muscle Cells 16%
• Human Aortic Smooth muscle Cells (HASMCs) 16%
• Matrix Metalloproteinase Activity 16%
• Airway Smooth muscle Cell Proliferation 16%
• Gene Expression 8%
• Western Blotting 8%
• Protein Expression 8%
• Molecular Mechanism 8%
• Airway 8%
• Estrogen Signaling 8%
• Estrogen Receptor Signaling 8%
• Muscle Mass 8%
• Mass Matrix 8%
• Pathway Activity 8%
• Luciferase Reporter Assay 8%
• Quantitative RT-PCR 8%
• Extracellular Matrix Genes 8%
• FASEB 8%
• Matrix Gene Expression 8%
• Airway Physiology 8%

Pharmacology, Toxicology and Pharmaceutical Science

• Estrogen Receptor 100%
• Asthma 36%
• Tumor Necrosis Factor 27%
• Platelet Derived Growth Factor 27%
• Receptor Agonist 18%
• Airway Remodeling 18%
• Transcription Factor AP 1 18%
• 1,3,5 Tris(4 Hydroxyphenyl) 4 Propylpyrazole 18%
• Matrix Metalloproteinase 18%
• Western Blot 9%
• Luciferase 9%

Biochemistry, Genetics and Molecular Biology

• Estrogen Receptor 100%
• Platelet-Derived Growth Factor 27%
• Tumor Necrosis Factor 27%
• AP-1 Transcription Factor 18%
• Receptor Agonists 18%
• Matrix Metalloproteinase 18%
• Quantitative Reverse Transcription Polymerase Chain Reaction 9%
• Western Blot 9%
• Gene Expression 9%
• Cell Proliferation 9%
• Luciferase 9%