Dietary Nutrients and Prevention of Alzheimer’s Disease

Pallavi Singh Chauhan, Dhananjay Yadav, Ananta Prasad Arukha

Research output: Contribution to journalReview articlepeer-review

8 Scopus citations


Alzheimer’s disease is an irrevocable, progressive brain disorder that gradually destroys memory and cognitive skills. One of the extensively studied methods of preventing Alzheimer’s disease (AD) progression is by providing a nutritional diet. Several reports have shown that intake of nutritional elements as huperzine A, ursolic acid, vitamins etc., can directly influence pathogene-sis of AD. Surprisingly, the occurrence of metabolic disorders due to an unhealthy diet has been known to be a major environmental cause of AD. It has been noted that AD severity can be controlled by supplementing dietary supplements containing huge amounts of health-promoting ingre-dients. These elements promote cell health, regeneration, and the anti-aging process that specifical-ly interrupt the pathogenic pathways in AD development. Fortunately, incorporating changes in the nutritional content is inexpensive, easy, acceptable, safe, effective, and in most cases, free from major adverse events. Many nutritional phytoconstituents such as flavonoids, alkaloids, and terpenoids are still being evaluated in the hope of identifying a successful therapy for AD. This review discusses the therapeutical potential of several key nutrients that have been researched for treating AD treatment and the method of their neuroprotective intervention.

Original languageEnglish (US)
Pages (from-to)217-227
Number of pages11
JournalCNS and Neurological Disorders - Drug Targets
Issue number3
StatePublished - Apr 2022
Externally publishedYes

Bibliographical note

Funding Information:
This work was supported by the National Research Foundation of Korea (NRF) funded by the Ministry of Education (NRF-2019R1G1A1008566) South Korea.

Publisher Copyright:
© 2022 Bentham Science Publishers.


  • Alzheimer’s Disease
  • Brain disease
  • Inflammation
  • Neurodegeneration
  • Nutrition
  • Oxidative stress

PubMed: MeSH publication types

  • Journal Article
  • Research Support, Non-U.S. Gov't
  • Review


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