Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: The effects of CaMKII and angiotensin AT1 receptor inhibition

Tomas Rajtik, Slavka Carnicka, Adrian Szobi, Zoltan Giricz, Jin O-Uchi, Veronika Hassova, Pavel Svec, Peter Ferdinandy, Tanya Ravingerova, Adriana Adameova

Research output: Contribution to journalArticlepeer-review

3 Scopus citations

Abstract

Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" [1].

Original languageEnglish (US)
Pages (from-to)730-734
Number of pages5
JournalData in Brief
Volume7
DOIs
StatePublished - Jun 1 2016
Externally publishedYes

Bibliographical note

Funding Information:
This work was supported by grants VEGA 1/0638/12 , APVV 0102-11 and by Hungarian Scientific Research Fund ( OTKA K 109737 ). ZG holds a “János Bolyai Fellowship” from the Hungarian Academy of Sciences. PF is a Szentágothai Fellow of the National Program of Excellence (TAMOP 4.2.4.A/2-11-1-2012-0001).

Keywords

  • AT receptor blockade
  • Apoptosis
  • CaMKII inhibition
  • Myocardial ischemia/reperfusion injury
  • Necrosis

Fingerprint Dive into the research topics of 'Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: The effects of CaMKII and angiotensin AT<sub>1</sub> receptor inhibition'. Together they form a unique fingerprint.

Cite this