Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" .
Bibliographical noteFunding Information:
This work was supported by grants VEGA 1/0638/12 , APVV 0102-11 and by Hungarian Scientific Research Fund ( OTKA K 109737 ). ZG holds a “János Bolyai Fellowship” from the Hungarian Academy of Sciences. PF is a Szentágothai Fellow of the National Program of Excellence (TAMOP 4.2.4.A/2-11-1-2012-0001).
- AT receptor blockade
- CaMKII inhibition
- Myocardial ischemia/reperfusion injury