Abstract
Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" [1].
Original language | English (US) |
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Pages (from-to) | 730-734 |
Number of pages | 5 |
Journal | Data in Brief |
Volume | 7 |
DOIs | |
State | Published - Jun 1 2016 |
Externally published | Yes |
Bibliographical note
Publisher Copyright:© 2016 The Authors.
Keywords
- AT receptor blockade
- Apoptosis
- CaMKII inhibition
- Myocardial ischemia/reperfusion injury
- Necrosis