Skip to main navigation
Skip to search
Skip to main content
Experts@Minnesota Home
Home
Profiles
Research units
University Assets
Projects and Grants
Research output
Press/Media
Datasets
Activities
Fellowships, Honors, and Prizes
Search by expertise, name or affiliation
Cutting edge: Inflammatory signals drive organ-specific autoimmunity to normally cross-tolerizing endogenous antigen
Vaiva Vezys
, Leo Lefrançois
Microbiology
Research output
:
Contribution to journal
›
Article
›
peer-review
65
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Cutting edge: Inflammatory signals drive organ-specific autoimmunity to normally cross-tolerizing endogenous antigen'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Ag Release
50%
Autoimmunity
50%
Causative Agents
50%
CD40
50%
CD8+ T Cells
50%
Cholera Toxin
50%
Concomitant Infection
50%
Cytokine Release
50%
Enterocyte
50%
Icell
50%
Inflammatory Pathways
100%
Intestinal Diseases
50%
Intestine
50%
Lymph Node
50%
Organ-specific Autoimmunity
100%
OT-I
100%
Self-protein
50%
T Cell Recognition
50%
T Cells
50%
Tissue Damage
50%
Tolerogenic
50%
Transgenic
50%
Transgenic Mice
50%
Vesicular Stomatitis Virus
100%
Immunology and Microbiology
Antigen-Presenting Cell
66%
Autoimmunity
100%
Cholera Toxin
33%
Cytokine Release
33%
Cytotoxic T-Cell
33%
Infectious Agent
33%
Intestine
33%
Lymph Node
33%
Self-Protein
33%
T Cell
33%
Transgenic Mouse
33%
Transgenics
33%
Vesiculovirus
66%
Wild Type
33%