The present study is to examine an alternative mechanism for compressively-induced crack growth and to compare it quantitatively to previous studies. Immediately following implantation, the interface contains imperfections and gaps. These gaps, due to their opposition to bone, will tend to fill with interstitial fluid. If these fluid-filled voids are sealed off from the porous bone by fibrous tissue growth or bone remodelling, they may become pressurized upon compressive loading of the interface. The purpose of the present work is to determine whether it is feasible to propagate mode I cracks through this mechanism. To answer this question, a two-dimensional crack problem is solved.
|Original language||English (US)|
|Number of pages||4|
|Journal||American Society of Mechanical Engineers, Applied Mechanics Division, AMD|
|State||Published - Jan 1 1987|