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Breast Cancer Progression by the FGF/FGFR Axis: A Metabolic Perspective
Jennifer E. Tuokkola
,
Kathryn L. Schwertfeger
Laboratory Medicine and Pathology
Research output
:
Contribution to journal
›
Review article
›
peer-review
4
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Scopus citations
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Keyphrases
Metabolic
100%
Breast Cancer Progression
100%
Fibroblast Growth Factor Receptor 1 (FGFR1)
100%
Breast Cancer
80%
Fibroblast Growth Factor Receptor Signaling
80%
Transcription Factor
20%
Biomarker Development
20%
Tissue Repair
20%
Lipid Metabolism
20%
Tumor Progression
20%
Tumor Metastasis
20%
Dysregulated
20%
Chemotherapy Resistance
20%
Intervention Development
20%
Metabolic Homeostasis
20%
Metabolic Plasticity
20%
Receptor-mediated
20%
Nutrient Sensing
20%
Metabolic Function
20%
Metabolic Enzymes
20%
Tumor Therapeutics
20%
Tumor Heterogeneity
20%
Cellular Signaling
20%
OR Genes
20%
Tumor Resistance
20%
Gene Fusion
20%
Receptor Activity
20%
Subtype-specific
20%
Multiple Cancer Types
20%
Glycolytic Metabolism
20%
Isoform Switching
20%
Metabolic Vulnerability
20%
Therapeutic Biomarkers
20%
Resistance to Targeted Therapy
20%
Cancer Cell Metabolism
20%
Therapeutic Interventions
20%
Metabolic Pathways
20%
Biochemistry, Genetics and Molecular Biology
Fibroblast Growth Factor Receptor
100%
Signal Transduction
11%
Isoform
11%
Lipid Metabolism
11%
Cancer Cell
11%
Enzyme
11%
Homeostasis
11%
Mediator
11%
Gene Fusion
11%
Cell Metabolism
11%
Tissue Repair
11%
Transcription Factors
11%
Tumor Progression
11%
Targeted Therapy
11%