Atrial fibrillation associated with hypokalemia due to primary hyperaldosteronism (Conn's syndrome)

Basel Al Aloul, Jian-Ming Li, David G Benditt, Venkatakrishna N Tholakanahalli

Research output: Contribution to journalArticle

10 Citations (Scopus)

Abstract

Aldosterone is a major regulator of the body potassium stores through its role in potassium excretion by the kidney. However, it is unclear whether aldosterone affects transcellular distribution of potassium in atrial myocytes, and whether hypokalemia associated with primary hyperaldosteronism increases susceptibility to atrial fibrillation (AF). We report a case of a 58-year-old male with Conn's syndrome in whom symptomatic AF was related to hypokalemia.

Original languageEnglish (US)
Pages (from-to)1303-1305
Number of pages3
JournalPACE - Pacing and Clinical Electrophysiology
Volume29
Issue number11
DOIs
StatePublished - Nov 1 2006

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Hyperaldosteronism
Hypokalemia
Atrial Fibrillation
Potassium
Aldosterone
Muscle Cells
Kidney

Keywords

  • Atrial fibrillation
  • Hyperaldosteronism
  • Hypokalemia

Cite this

Atrial fibrillation associated with hypokalemia due to primary hyperaldosteronism (Conn's syndrome). / Aloul, Basel Al; Li, Jian-Ming; Benditt, David G; Tholakanahalli, Venkatakrishna N.

In: PACE - Pacing and Clinical Electrophysiology, Vol. 29, No. 11, 01.11.2006, p. 1303-1305.

Research output: Contribution to journalArticle

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