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Ataxin-1 with an expanded glutamine tract induces disease via alterations in nuclear function
H. Orr
, I. Skiement
,
P. Skinner
, J. Davidson
, E. Burright
, C. Cumminigs
, B. Koshy
, H. Zoghh
Laboratory Medicine and Pathology
Host-Pathogen Interface Biology
Genetics Mechanisms of Cancer
Research output
:
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Article
›
peer-review
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Keyphrases
Associated Proteins
10%
Ataxia
50%
Ataxin-1
100%
COS Cells
20%
Glutamine
100%
Hybrid Study
10%
Matrix-based
10%
Molecular Model
10%
Mouse Genetics
10%
Neurogenetic Disorders
10%
Nuclear Distribution
20%
Nuclear Function
100%
Nuclear Inclusions
10%
Nuclear Localization Sequence
10%
Nuclear Proteins
10%
Polyglutamine Tract
10%
Purkinje Cell
10%
Self-association
10%
Similarity Structure
10%
Yeast Two-hybrid
10%
Neuroscience
Ataxia
62%
Ataxin
25%
Ataxin 1
100%
COS Cells
25%
Glutamine
100%
Molecular Model
12%
Nuclear Localization Signal
12%
Nuclear Protein
12%
Polyglutamine
12%
Purkinje Cell
12%
Vitamin A
12%