Aryl hydrocarbon receptor-dependent inhibition of AP-1 activity by 2,3,7,8-tetrachlorodibenzo-p-dioxin in activated B cells

Keyphrases

• Activator protein-1 (AP-1) 100%
• Activated B Cells 100%
• 2,3,7,8-tetrachlorodibenzo-p-dioxin 100%
• Aryl Hydrocarbon Receptor 100%
• B Cells 50%
• CH12 40%
• Lipopolysaccharide 20%
• Lipopolysaccharide-induced 20%
• C-Jun 20%
• Nuclear factor-B 20%
• Gene Expression 10%
• MRNA Expression 10%
• B Cell Activation 10%
• Cellular Targets 10%
• Transcriptional Activity 10%
• Humoral Immunity 10%
• Steady State 10%
• Major Components 10%
• Electrophoretic Mobility Shift Assay 10%
• Supershift 10%
• DNA Binding 10%
• Reporter Gene 10%
• Cellular Activation 10%
• Chloramphenicol Acetyltransferase 10%
• AP-1 Complex 10%
• Tetrachlorobiphenyl 10%
• AP-1 Factors 10%
• AhR Antagonist 10%
• α-Naphthoflavone 10%
• Immunoglobulin Gene Expressions 10%

Pharmacology, Toxicology and Pharmaceutical Science

• 2,3,7,8-Tetrachlorodibenzodioxin 100%
• Aromatic Hydrocarbon Receptor 100%
• Lipopolysaccharide 40%
• Alternative Complement Pathway C3 C5 Convertase 20%
• Nuclear Factor 20%
• Immunoglobulin M 20%
• Immunoglobulin 10%
• Chloramphenicol Acetyltransferase 10%
• Electrophoretic Mobility Shift Assay 10%
• 2,3',4',5 Tetrachlorobiphenyl 10%
• Messenger RNA 10%