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Apigenin Decreases Acinar Cell Damage in Pancreatitis
Amy A. Mrazek
, Vandanajay Bhatia
, Miriam Falzon
, Heidi Spratt
, Celia Chao
, Mark R. Hellmich
Research output
:
Contribution to journal
›
Article
›
peer-review
7
Scopus citations
Overview
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Keyphrases
Pancreatitis
100%
Acinar Cells
100%
Cell Damage
100%
Apigenin
100%
Recurrent Acute Pancreatitis
62%
Parathyroid Hormone-related Protein (PTHrP)
62%
Transforming Growth Factor-β
37%
Nuclear Localization
37%
Mouse Model
25%
Messenger RNA (mRNA)
25%
Interleukin-6
25%
Inflammatory Pathways
25%
Tumor Necrosis Factor-α
25%
Promoter Activity
25%
Nuclear factor-B
25%
Signaling Pathway
12%
Chronic Pancreatitis
12%
Immunohistochemistry
12%
Attenuation
12%
Knock-in Mouse Model
12%
Wild Mice
12%
Inflammatory Response
12%
MRNA Level
12%
Immunoblotting
12%
ERK Pathway
12%
Acinus
12%
Pathway Activity
12%
Protein Factor
12%
Multiple Episodes
12%
PD98059
12%
ERK MAPK
12%
AR42J Cells
12%
Cell Architecture
12%
Dual-luciferase Reporter Assay
12%
Medicine and Dentistry
Pancreatitis
100%
Cell Damage
100%
Acinar Cell
100%
Apigenin
100%
Parathyroid Hormone-Related Protein
62%
Acute Pancreatitis
62%
Cancer Growth Factor
37%
In Vitro
25%
Tumor Necrosis Factor
25%
Alternative Complement Pathway C3 C5 Convertase
25%
Nuclear Factor
25%
Ceruletide
25%
Interleukin 6
25%
Messenger RNA
25%
Serositis
12%
Cell Signaling Pathway
12%
Sequela
12%
Immunohistochemistry
12%
Promoter Region
12%
Acinus
12%
2 (2 Amino 3 Methoxyphenyl)chromone
12%
Luciferase Assay
12%
Promoter Protein
12%
Chronic Pancreatitis
12%
Mitogen-Activated Protein Kinase
12%
Immunoblotting
12%
Knockout Mouse
12%