A eukaryotic-type Ser/Thr kinase in Enterococcus faecalis mediates antimicrobial resistance and intestinal persistance

Keyphrases

• E. Faecalis 100%
• Antimicrobial Resistance 100%
• Enterococcus Faecalis 100%
• Persistance 100%
• Antibiotics 33%
• Signaling Molecules 33%
• Bile 33%
• Kinase Domain 33%
• One-component 33%
• Nosocomial Pathogens 33%
• Linezolid Resistant Enterococci 33%
• Genetic Determinants 16%
• Novel Therapies 16%
• Cell Wall 16%
• Physiological Processes 16%
• Intestinal Tract 16%
• Hospital-acquired Infection 16%
• Cell Wall Biogenesis 16%
• Intestine 16%
• Transmembrane Segment 16%
• Peptidoglycan 16%
• Enhanced Sensitivity 16%
• Detergent 16%
• Adaptive Response 16%
• Nosocomial Infection 16%
• Active Compounds 16%
• Extracellular Domain 16%
• Membrane Receptors 16%
• Cell Wall Integrity 16%
• Cell Envelope 16%
• Domain-specific Architecture 16%
• Intestinal Persistence 16%
• Inherent Resistance 16%
• Antimicrobial Stress 16%
• Preventing Infection 16%

Biochemistry, Genetics and Molecular Biology

• Enterococcus faecalis 100%
• Phosphotransferase 100%
• Kinase 100%
• Antibiotic Resistance 100%
• Signaling Protein 66%
• Infectious Agent 66%
• Enterococcus 66%
• Genetics 33%
• Wild Type 33%
• Biogenesis 33%
• Adaptive Response 33%
• Physiological Process 33%
• Transmembrane Receptors 33%
• Target Cell 33%
• Peptidoglycan 33%

Immunology and Microbiology

• Cell Wall 100%
• Enterococcus faecalis 100%
• Infection 100%
• Antibiotic Resistance 100%
• Infectious Agent 66%
• Enterococcus 66%
• Biogenesis 33%
• Intestine 33%
• Target Cell 33%
• Physiological Process 33%
• Wild Type 33%
• Peptidoglycan 33%