5. THE ROLE OF BILE ACIDS IN THE MODULATION OF APOPTOSIS

Keyphrases

• Apoptosis 100%
• Bile Acids 100%
• Tauroursodeoxycholic Acid (TUDCA) 85%
• Liver Disease 28%
• Anti-apoptotic 28%
• Ischemic Stroke 14%
• Induced Apoptosis 14%
• Transgenic Mouse Model 14%
• Clinical Application 14%
• Ligand-independent 14%
• Phosphoinositide 3-kinase (PI3K) 14%
• Apoptotic Pathway 14%
• Cell Survival 14%
• Graft Function 14%
• Therapeutic Agents 14%
• Rat Model 14%
• Cell Death 14%
• Oral Administration 14%
• Cell Behavior 14%
• Acid Activation 14%
• Cell Function 14%
• Neurodegenerative Diseases 14%
• Mitochondrial Pathway 14%
• Water-soluble 14%
• Neurologic 14%
• Plasma Membrane 14%
• Cholestasis 14%
• NF-B 14%
• Cytochrome c Release 14%
• Cellular Structure 14%
• Hepatic Cells 14%
• Hepatocyte Injury 14%
• Insults 14%
• Caspase Activation 14%
• Survival Signaling 14%
• Neuronal Injury 14%
• Death Signals 14%
• Promising Drug 14%
• Apoptotic Mechanism 14%
• Animal Experiment 14%
• Fas Receptor 14%
• Detergent Effect 14%
• Bax Translocation 14%
• Hemorrhagic Stroke 14%
• Intracellular Signaling Molecules 14%
• Mitochondria-associated Membranes 14%
• Regulatory Property 14%
• Induced Hepatocytes 14%
• Permeability Transition Pore 14%
• Membrane Perturbation 14%
• Death Receptor Pathway 14%
• Rat Model of Parkinson's Disease 14%
• Substrate Cleavage 14%
• Damage Range 14%
• Toxic Molecules 14%
• Graft Survival 14%
• Caspase Substrates 14%

Pharmacology, Toxicology and Pharmaceutical Science

• Bile Acid 100%
• Rat Model 28%
• Antiapoptotic 28%
• Liver Disease 28%
• Transgenic Mouse 14%
• Mouse Model 14%
• Brain Ischemia 14%
• Brain Hemorrhage 14%
• Death Receptor 14%
• Cytoprotective Agent 14%
• Tumor Necrosis Factor Receptor Superfamily Member 6 14%
• Cholestasis 14%
• Parkinson's Disease 14%
• Caspase 14%
• Cytochrome C 14%
• Oral Administration 14%