Aim 1. To evaluate the role of dystrophin and sarcoglycan complex in the modulation of coronary flow in response to changes in endothelial shear stress and hormonal mediated vasoconstriction and vasodilation. Hypothesis: Vascular smooth muscle contracti
|Effective start/end date||4/1/10 → 3/31/15|
- NIH NAT'L HEART, LUNG, AND BLOOD INSTITU
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