Keyphrases
Spinocerebellar Ataxia
100%
Purkinje Cell
88%
Mouse Model
58%
Bergmann Glia
58%
Brain Diseases
58%
Single-nucleus RNA Sequencing
50%
Calcium Signaling
50%
Extracellular Matrix
50%
Transgenic
50%
Glial Changes
50%
Cerebellum
50%
Cell Changes
50%
Ataxin-1
33%
Microglia
22%
Extracellular Matrix Remodeling
16%
Hypoxia-inducible factor-1α (HIF-1α)
16%
Glial Cells
16%
Hyperexcitability
15%
Wild Mice
15%
Abnormal Expansion
12%
CAG Repeat
12%
Cerebellar Purkinje Cell
12%
Spontaneous Rate
11%
Perineuronal Nets
11%
Transcription Factor
11%
Cerebellar Ataxia
9%
Non-cell Autonomous
8%
Human Brain
8%
Normal Brain
8%
Early Disease
8%
Brain Function
8%
Glia
8%
Neuronal Pathology
8%
Pcp2
8%
Gene Alteration
8%
Cell Dysfunction
8%
Astrocytes
8%
Neuroprotective
8%
Oligodendrocyte
8%
Gliosis
8%
Reactive Gliosis
8%
Pathway Alteration
8%
Progressive Neurodegeneration
8%
Synaptic Properties
7%
Intrinsic Properties
7%
Hyperactivity
7%
Hypoxia Response
5%
Disease States
5%
Brain State
5%
Matrix-based
5%
Neuroscience
Spinocerebellar Ataxia
100%
Purkinje Cell
92%
Glia
66%
Encephalopathy
58%
Cerebellum
50%
Nuclear RNA
50%
RNA Sequence
50%
Extracellular Matrix
50%
Calcium Signaling
50%
Glial Cells
16%
Gliosis
16%
Microglia
15%
Neurodegenerative Disorder
12%
CAG Repeat
12%
Astrocyte
12%
Firing Rate
11%
Hypoxia Inducible Factor 1
11%
Neuroprotective Agent
8%
Brain Function
8%
Oligodendrocyte
8%
Perineuronal Net
7%
Gene Expression
7%
Hyperactivity
7%
Ataxia
7%
Medicine and Dentistry
Calcium Signaling
50%
Spinocerebellar Ataxia
50%
Glia
50%
Purkinje Cell
42%
Firing Rate
11%
Isotopes of Calcium
7%
Hyperactivity
7%
